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[1] Zheng, M., Zhang, X., Zhou, Y., et al. (2019). TCR repertoire and CDR3 motif analyses depict the role of alphabeta T cells in Ankylosing spondylitis. EBioMedicine 47: 414−426. DOI: 10.1016/j.ebiom.2019.07.032. [2] Zheng, M. (2023). Autoreactive T cells of ankylosing spondylitis elicited by COVID-19 infection: A snapshot of immunological host defense and autoimmune imprinting. Autoimmun. Rev. 22: 103392. DOI: 10.1016/j.autrev.2023.103392. [3] Britanova, O.V., Lupyr, K.R., Staroverov, D.B., et al. (2023). Targeted depletion of TRBV9(+) T cells as immunotherapy in a patient with ankylosing spondylitis. Nat. Med. 29: 2731−2736. DOI: 10.1038/s41591-023-02613-z. [4] Lerner, A., Jeremias, P., and Matthias, T. (2015). The world incidence and prevalence of autoimmune diseases is increasing. Int. J. Celiac. Dis. 3: 151−155. DOI: 10.12691/ijcd-3-4-8. [5] Klunk, J., Vilgalys, T.P., Demeure, C.E., et al. (2022). Evolution of immune genes is associated with the Black Death. Nature 611: 312−319. DOI: 10.1038/s41586-022-05349-x. -
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Zheng M. (2024). Autoimmune imprinting in the post-COVID-19 era. The Innovation Medicine 2(1): 100054. https://doi.org/10.59717/j.xinn-med.2024.100054
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Zheng M. (2024). Autoimmune imprinting in the post-COVID-19 era. The Innovation Medicine 2(1): 100054. https://doi.org/10.59717/j.xinn-med.2024.100054
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Figure 1.
Navigating the entanglement of immune defense and autoimmune inflammation: precision medicine on the horizon.
